RTN4/NoGo-receptor binding to BAI adhesion-GPCRs regulates neuronal developmentRTN4Rs, surface proteins expressed in neurons, interact with BAI adhesion-GPCRs located in either neurons or glia via a glycan-mediated interface, and this interaction regulates the dendritic arborization, axonal elongation, and formation of synapses in cultured human neurons.RTN4Rs, surface proteins expressed in neurons, interact with BAI adhesion-GPCRs located in either neurons or glia via a glycan-mediated interface, and this interaction regulates the dendritic arborization, axonal elongation, and formation of synapses in cultured human neurons.Jie Wang, Yi Miao, Rebecca Wicklein, Zijun Sun, Jinzhao Wang, Kevin M. Jude, Ricardo A. Fernandes, Sean A. Merrill, Marius Wernig, K. Christopher Garcia, Thomas C. Südhofhttps://secure.jbs.elsevierhealth.com/action/getSharedSiteSession?redirect=https%3A%2F%2Fwww.cell.com%2Fcell%2Ffulltext%2FS0092-8674%2821%2901232-0%3Frss%3Dyes&rc=0http://www.cell.com/cell/inpress.rssCellCell RSS feed.Wireless News CampaignNovember 10, 2021

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